AAS With a Little Help from AvantLabs

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MaxMuscle
Super Heavy Weight
2005/05/18 23:03:17 (permalink)
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AAS With a Little Help from AvantLabs

AAS With a Little Help from AvantLabs


by Prolangtum

I would like to introduce you to a group of drugs that are near and dear to my heart: Anabolic Androgenic Steroids.  This little article is going to give you a few ideas if you someday want to venture to the so-called "dark side" of bodybuilding.  As a disclaimer, I am not recommending you break the laws of your respective country and use AAS if they are indeed illegal where you are.  This may disappoint a few, but this is not going to be filled with references to a myriad number of studies.  That just is not my style.  I will leave that to the likes of Loki and Nandi.  I am neither witty nor clever; so do not expect that from my writing.  Nothing fancy, nothing sexy.  The only thing sexy about this article is the author.

I am also not a writer by trade.  So if you do not like my writing style, than I suggest you write your own damn article.  Now, to what this article is going to be.  It is going to be a basic outline of sample cycles pieced together from my experience with nearly every illicit anabolic steroid used commonly by bodybuilders.  You can speculate what works by sifting through abstract after abstract on pubmed, but the vast majority of the time, "Real World" experience trumps science. I am also not going to lay out use of anti-estrogens or post cycle therapy, as that would warrant another article in itself.  Now, let’s get down to it.
 
A Combo with Shades of Legality (But still Illegal)
 
Before I get into generic bulking and cutting cycles, I will offer a somewhat legal route many may choose to go. A cattle farmer can order Finaplix H or Synovex H cartridges without a prescription. But can Joe Juicer in downtown New York order them without a prescription? Yes, but being in possession of them opens yourself up to possible legal trouble. To date, I have not heard of anyone being arrested or charged with anything stemming from ordering Finaplix or Synovex carts. Turning them into injectables for human administration is definitely illegal. 

2 carts of Finaplix H and a conversion kit will cost roughly $100, and it will yield 4 grams of trenbolone acetate at the usual concentration of 75mg/ml. 5 carts of Synovex H and a conversion kit will cost roughly $130, and it will yield 8 grams of testosterone without an ester at the concentration of 100mg/ml. A loss of mgs is inevitable in the conversion process—I usually guesstimate an 85% yield.  One drawback of using these compounds in a first cycle is that you have to jump head first into everyday injections.  With the testosterone having no ester, it needs to be injected at least every day, some may say 2 times a day.  An example of a bulking cycle for your first time steroid user weighing 200 lbs:

8 weeks of TNE (Test no ester) @ 75mg ed
8 weeks of TA (trenbolone acetate) @ 100mg eod
Week 9 PCT (3 days after last injection)

Cutting cycles in my opinion should only use enough drugs in order to prevent muscle loss, so the dosages will be lower. Using our same 200 lb. bodybuilder:
       
8 weeks of TNE @ 50mg ed
        8 weeks TA @ 75mg eod
        Week 9 PCT (3 days after last injection)

After only a couple weeks of everyday injections even the most veteran steroid user will tire of the process. I myself have a weird fetish for needles and look forward to injecting, but that is neither here nor there. If you are looking to stay away from androgenic sides, then this combination is not for your.  Both compounds are very androgenic.  Testosterone will aromatize, so having nolvadex on hand is standard procedure.  Trenbolone does not aromatize, but is progesteronic.  Despite popular message board opinion, progesterone does not directly cause gynecomastia. But again, having nolvadex on hand is still key.   Trenbolone and Testosterone are a very potent combination yielding some dramatic body composition changes in either bulking or cutting cycles and the duo offers great bang for the buck.

Bulking Cycles

Now to the meat and potatoes of steroid use: The Bulking Cycle.  If you have never touched a steroid, I would suggest you venture here first.  As a general rule of thumb, your first cycle is the best cycle, so why waste all that potential lean body mass gain from your first cycle by using steroids to "cut".  Besides as we all know, cutting blows.

My first suggestion to a first time steroid user is a simple but effective cycle of just Testosterone with a long acting ester such as Enanthate (14 day half life) or Cypionate (12 day half life).  Testosterone has a multitude of benefits, and it should be part of every cycle in my opinion.   A dose of 500mgs will be plenty for any beginner or advanced steroid user for that matter.  If you’re wanting to "kickstart" the cycle, I would suggest throwing in Testosterone Propionate (3 day half life) for the first week to get blood levels up immediately. A short ester such as Propionate is also helpful as the cycle winds down, as one is waiting for the Enanthate/Cypionate to clear for post cycle therapy.  Again using our 200 lb. bodybuilder I would lay it out as follows:

10 weeks of Testosterone Enanthate/Cypionate @ 500mgs per week
2 weeks (Week 1 + 11) of Testosterone Propionate @ 150mgs eod
Week 12 PCT

A more advanced user or someone who is adventurous could stack another injectable in their first cycle.  Both Nandrolone Deconate (aka Deca) and Boldenone Undecylenate (aka Equipoise or Eq) are relatively mild injectables that have fairly low side affects and are very anabolic.  An example would be as follows:

10 weeks of Testosterone Enanthate/Cypionate @ 500mgs per week
3 weeks (Week 1 + 11,12) of Testosterone Propionate @ 150mgs eod
10 weeks of Deca @ 300mgs per week or 10 weeks of Eq @ 400mgs per week
Week 13 PCT

I generally recommend that first time users stay away from orals, but some may choose to use them in their cycle More likely an advanced user will want to incorporate them.  Check out  Hepatoxicity: Fact or Fiction and decide for yourself what stance you wish to take on oral steroids.  Methandrostenolone (aka Dbol) or Oxymetholone (aka Anadrol) are two oral steroids used for bulking.  Water retention and estrogenic sides are very likely with these compounds, but the tradeoff is massive strength and weight gains. Using the base model cycles I have proposed, one would incorporate them as following:

10 weeks of Testosterone Enanthate/Cypionate @ 500mgs per week
1 week (11) of Testosterone Propionate @ 150mgs eod
4-6 weeks of Dbol @ 35mgs ed or 4-6 weeks of Anadrol @ 50-75mgs ed
Week 12 PCT

The final bulking cycle I will propose will include a somewhat newer combination of an ester and hormone.  Many underground labs have introduced Trenbolone Enanthate in the past couple of years.  You get all the benefit of Trenbolone with the added bonus of fewer injections. This will be a 3 compound cycle, although advanced users may want to throw in an oral steroid in the beginning.  If so, I would drop the Test Propionate.

10 weeks of Testosterone Enanthate/Cypionate @ 500mgs per week
2 weeks (1+11) of Testosterone Propionate @ 150mgs eod
10 weeks of Trenbolone Enanthate @ 400-600mgs per week
Week 12 PCT

Cutting Cycles
 
The phrase "cutting cycles" is a bit misleading.  Although some steroids have shown to help change body composition, steroids have no real thermogenic properties.  Anabolic Steroids do however have a dramatic impact on your body chemistry, making altering body composition much easier.  That being said, the amount of drugs in my opinion should be less than a bulking cycle.  The main goal is to use just enough drugs to offset any diet induced muscle loss.  Your diet and training are going to do the legwork when cutting.  I also want to address the misconception that there are "cutting steroids" and "bulking steroids".  You can cut on Dianabol or bulk on Winstrol.  But, some drugs might be considered more favorable towards one goal or the other because of their impact on water retention or lack there of. 

Adhering to the K.I.S.S. acronym, the first cycle is going to be simple, but it is very effective.  Dieting is not cheap, and by using an inexpensive compound like Testosterone only, you can save your money for other things such as food, thermogenics and appetite suppressants.  Dieting tends to put one's mood and libido in the toilet.  A nice dose of Testosterone works as a mild anti depressant and makes the dog look sexy.  What more could you want?  Using our same 200 lb. bodybuilder as an example my suggested cycle would be:

8 weeks of Testosterone Propionate @ 100mgs eod
Week 9 PCT (3 days after last injection)

The next suggested cycle is going to add a drug that is not my favorite by any means, but is very effective.  We will add Winstrol to the Testosterone Propionate (Test Prop. from now on) at a mild dose.  This drug is renowned for the hardening affect it seems to have on muscles, as well as generating increases in strength, even when dieting.

There are however a couple of reasons why I do not like it.  After one week of use, all of my joints ache to the point that lifting heavy is not an option for me.  Those prone to Male Pattern Baldness will notice some thinning of the hair as well (I fall into that category).  But, take note that I was using 100mgs ed.  People are different, so some may not see these side effects even at that large of a dose.  A saner dose will minimize or eliminate those sides.  Being orally bioavailable, one can skip injecting the Winstrol even if it is in a water-based suspension.  Keeping moderation in mind:

8 weeks of Test Prop. @ 100mgs eod
8 weeks of Winstrol @ 25mgs ed
Week 9 PCT (3 days after last injection)

The last cutting cycle I will suggest has been deemed by some as the ultimate dieting stack.  I will not go that far, but I will say it is a very potent combination.  Although I have touched on Trenbolone before in the first section, I am bringing it back for the last cycle.  Some on the internet have reported Trenbolone's almost "magic" effect of drastically altering body composition, even when not in a calorie deficit.  Although all steroids will have an effect on body composition, Trenbolone's seems to be the most pronounced. 

The combination of Test Prop, Winstrol and Trenbolone is going to be the most potent stack of all, but with that, it will also yield the most side effects.  There are no free rides when it comes to steroids: you have to pay the piper eventually.  It is very androgenic, and the possibilities of hair loss, increased acne and prostate problems are the greatest on this stack.  You have to make the intelligent decision regarding what you are willing to accept as consequences.  I would suggest this to only the most advanced user, or one who is getting ready for a contest.  If you are just in it for aesthetics, it is my opinion that you should not use this cycle.  I am not one for moderation, but even I know what compounds I am just not going to stack.  Do not get idea that this stack is going to make you die post-injection.  I just do not want people blaming me for going bald in their early 20s. With the addition of Trenbolone the cycle will look like:

8 weeks of Test Prop. @ 100mgs eod
8 weeks of Winstrol @ 25mgs ed
8 weeks of Trenbolone @ 100mgs eod
Week 9 PCT (3 days after last injection)

The Inclusion of Avant Lab Products

Unfortunately the bodybuilding community has a misguided mindset.  For the most part, more is always better and if it is not illegal, it is not worth buying.  I was once lost in this mindset for a few years.  I have since seen the light.  If you include one and only one legal supplement in your supplement arsenal while on cycle, I would suggest SesaThin.  The informed bodybuilder should already be taking this on a daily basis 365 days of the year.  For a further in depth review of all the benefits of using SesaThin while partaking in androgens, please read Par Deus's SeasThin and Androgens  Just a quick overview of its benefits on cycle: it reduces or preventing Visceral Adipose Tissue accumulation, exerts a positive effect on cholesterol levels, and protects the liver. 

When bulking, I will suggest you use 1-2 servings daily.  Someone who is cutting will definitely benefit from the recommended 3 daily servings.  When bulking, fat gain is a necessary evil.   I have experimented recently with 4 servings daily and have been pleasantly surprised to say the least.  Let me set up the situation.  I have been using a large amount of Testosterone and eating an upwards of 5100 calories daily.  Since implementing 4 servings of SesaThin for roughly one month, not only has fat gain been significantly reduced, but my waistline has actually shrunk 1/2 an inch. 

Before you call B.S., let me explain.  My subcutaneous fat has not been reduced at all.  But after measuring and re-measuring, the 1/2 inch loss is legit.  This leads me to believe that I have experience a reduction in Visceral Adipose Tissue (VAT).  So not only is there science to back up SesaThin's benefits, I have experienced first hand evidence that it works well for reducing VAT.  I can only imagine what it would do when I am dieting.  As for the other Avant Labs products to use when on-cycle, they fall in the same recommendations when not using steroids.  LeptiGen Mass and possibly PhenoGen are two worthy additions when bulking.  When cutting, one could use LeptiGen Basic or Rebirth depending on body type and body fat level.  Steroids cause some to have an increased appetite, so when cutting it would be prudent to use H.E.A.T. Stack as well as LeptiGen Rebirth.

Some Final Thoughts

Because this is very important, I want to reiterate that I am not recommending anyone break the laws of their countries and use steroids illegally.  Even if they are legal in your country, I am not recommending you use them either.  If you are already reading Mind and Muscle, then I will make the assumption you are an informed and intelligent individual.  One must use his intelligence when deciding to use steroids.  This article was intended to act as a basic framework for prospective developing prospective cycles based on different goals.  Hopefully you do not use this as your sole base of research.  It is definitely not an all-encompassing piece.  Please do yourself a favor and put in time researching the possible side effects and how to counter them before making any decisions on steroid usage.




#1

3 Replies Related Threads

    MaxMuscle
    Super Heavy Weight
    RE: AAS With a Little Help from AvantLabs 2005/05/18 23:06:08 (permalink)
    0
    Hepatoxicty: Fact or Fiction


    by Roy Harper

    We all know that the alpha alkylated steroids are hepatotoxic, right….. But, is there actually any truth to this? We’ve been told for years that if you take 17 alpha-alkylated steroids, you will eventually run into liver problems. Never combine 17 aa’s, never go beyond 50mg day, never go longer than 4 weeks, etc. All of this is crap! As I we walk you through some studies, today, you’ll see 17 alpha-alkylated steroids can be hepatotoxic but not to the degree you would think.

    To make a steroid hepatotoxic, you need only a small change to a steroid molecule; A strong bond that cannot readily be down broken by enzymes in the liver. This may be a bond at the 17th position, or even at the 1st position (as in methenolone or proviron). Because the liver cannot easily break the steroid down before it is released in to the blood stream, this also results in the steroid to becoming more orally bio-available.

    We can see that the liver has to work harder to break down these steroids. Enzymes in the blood and tissue easily metabolize other steroids such as Testosterone. Commonly, this increase in liver activity has been viewed as a harmful process, but as you will see, this increase is, in and of itself, irrelevant. The liver is THE filter of the human body -- it can figure out what to do with just about anything. The only real problem comes in when one keeps their liver at full blast for long periods of time.

    Let’s look at some studies showing the hepatotoxicity of steroids. Here's one of my favorites, a study published in 1979[1]. Essentially, researches did a study of deaths caused by hepatic angiosarcoma (a malignant tumor of vascular tissue in the liver) between 1964 and 1974. Researchers found 131 reported cases of death from hepatic angiosarcoma. Out of the 131 cases, 3.1% (4 cases) were reported to be at all related to the use of androgenic-anabolic steroids. Keep in mind that these 4 people could have liver complications before any steroids were used, aka a genetic disposition. In fact there is no proof, in this study at least, that the anabolic-androgenic steroids even caused the hepatic angiosarcoma.

    This is the classic case of associating a cause with an effect, without any evidence, aside from both existing. Furthermore, based on the above numbers, there are only 0.4 cases of hepatic angiosarcoma reported each year, by those using AAS. Now consider the number of people on steroids at this time. Now factor in all the people that don’t know their ass from a hole in the ground when it comes to using AAS, properly. Clearly, this is very week evidence. Lastly there has not been a real increase in hepatic angiosarcoma since the early seventies. Meanwhile, there has been a huge, almost exponential, increase in steroid use during this period.

    Another study, that somewhat supports the previous hepatotoxicity case, showed the possibilities of hepatic adenomas(cysts in the liver) caused by androgenic-anabolic steroids[2]. In this study, a Japanese girl was found to have multiple liver lesions after the use of the drug oxymetholone (aka Anadrol). Most everyone “knows” that Anadrol is linked with liver problems, but a closer inspection into this study shows more.

    Apparently, this girl, starting at the age of 14, was diagnosed with aplastic anemia. She was prescribed oxymetholone at 30mg per day. This continued for 6 years until the lesions first appeared. Assuming that the girl was most likely around 100 lbs., this was a pretty heavy dosage. If you extrapolated this data out to a 200 - 250lbs. male, that would be taking approximately 60 - 90mg of anadrol per day for 6 years. Ouch!

    The researchers also stated that there were only 17 other cases of hepatic adenomas, found in English literature between 1975 and 1998. They failed to mention the causes of these 17 cases, but there is no reason to believe they were all using 17-AA androgens and 17 is certainly miniscule compared to the number of people who have used them. The authors’ finish off the study by saying the following: "This report may be helpful in identifying the population who is at risk of developing hepatic sex hormone-related tumors." So remember, if you're a small 14-year-old girl taking 30mg of Anadrol per day for 6 years, you may be at risk!

    Let's move on to some more useful studies. Take for example a 1995 study that showed the toxic effects of anabolic-androgenic steroids in primary rat hepatic cell cultures[3]. In this study the researchers used the following drugs and dosages:


    As proof of the hepatoxicity they used Lactate dehydrogenase release, neutral red retention, and glutathione depletion to determine plasma membrane damage, cell viability, and possible oxidative injury, respectively.

    What they showed was that the 17 alpha-alkylated steroids, methyltestosterone, stanozolol and oxymetholone, significantly increased Lactate dehydrogenase release and decreased neutral red retention at the 1x10^-4M dosage for 24h. Both methyltestosterone and oxymetholone also showed depleted glutathione at the 1x10^-4M dosage after 2h, 6h and 8h treatments. In other words they increased liver activity. You may also note that the other, non-alkylated steroids showed no significant difference in any levels. All in all this not only shows that 17 alpha-alkylated steroids are directly “hepatotoxic”, but also non-alkylated steroids are note hepatotoxic at all. But is this a real measure of hepatotoxicity? There is yet to be any correlation between the increase of the above-mentioned measurement and “hepatotoxicity”. Obviously, high dosages of the 17 alpha-alkylated steroids are potentially dangerous, but upon closer inspection, the study reveals more.

    Take a look, the researchers took cell cultures from the liversse of 60-day-old Sprague-Dawley rats. Not only are rat livers much smaller than human livers, but these were merely cultures. Furthermore, it was the 1x10^-4M concentrations that caused the most changes, but these are approximately 1 to a 1/3 of a full, daily human dosage -- at least for the 17 alpha-alkylated steroids. Even at the 1x10^-6M concentration, there were no significant changes observed. It's apparent that the levels of 17 alpha-alkylated steroids used were potentially toxic, but for a human to take the same amount would be insane. I'm guessing this could translate to maybe 4 grams every 24 hours or 28 grams a week if not more.

    What is common so far is we can only prove that any steroid, that is believed to be hepatotoxic, only increases liver activity. I’ll say it again, where is the correlation to hepatotoxicity? We know that if the liver is running at 100% for long periods this may cause complications, but this is akin to any other chemical, which is metabolized by the liver. Ever noticed that liver cancer due to alcoholism takes decades of constant alcohol abuse? It’s apparent that the possibility for hepatotoxicity is there, but for the smart steroid user this is nearly an impossible task.

    Another study done in 1999, attempted to show the acute and chronic effects of stanozolol on the liver[4]. In acute treatments of stanozolol, dosages not mentioned, both cytochrome P456 and b5 (microsomal enzymes) levels dropped after 48 hours, and then at 72 hours, levels significant increased. On the other hand, with chronic treatments, time or dosage not mentioned, these microsomal enzymes showed a decrease in levels. Researchers showed that both acute and chronic treatments resulted in "slight to moderate inflammatory or degenerative lesions in centrilobular hepatocytes", but the authors did not note true hepatotoxicity.

    How about we look at the other side of the story, the good studies. For instance, in a 1999 study, which looked at the effects of an 8-week cycle of 17 alpha-alkylated steroids[5]. The researchers used fluoxymesterone, methylandrostanolone, or stanozolol on rats at 2mg/kg-body weight, five times a week for 8 weeks. That's 182mg per dosage, for a 200lb man, or 910mg per week. Half of the rats were sedentary and the other were trained on a treadmill.

    Levels of NADH-cytochrome c reductase, succinate cytochrome c reductase, and cytochrome oxidase (showing liver activity), increased in the steroid-administered rats, while citrate synthase showed no change. Comparatively, in vitro, the "cytochrome oxidase and citrate synthase activities were insensitive to the AAS, whereas NADH-cytochrome c reductase and succinate cytochrome c reductase activities were partly inhibited."

    Furthermore, in vivo, each rat had liver enzyme levels that were within normal range. From this, the researchers determined that the steroid-administered rats, trained or sedentary, did not show "...classical serum indicators of hepatic function". Extrapolating this, 910mg a week for 8 weeks could potentially have little to no effect on the liver in humans.

    As for human studies, in 1999 researchers tried to prove that the hepatotoxicity of steroids is overstated[6]. In this study, 15 of the participants were bodybuilders using self-administered steroid dosages and 10 were non-steroid bodybuilders. Serum data was compared to 49 patients with viral hepatitis, and 592 exercising and non-exercising medical students. [

    All of the bodybuilders showed increases in aspartate aminotransferase (AST), alanine aminotransferase (ALT) and creatine kinase (CK) while gamma-glutamyltranspeptidase (GGT) levels were in the normal range. In comparison, hepatitis patients showed increased ALT, AST, and GGT levels while the control exercising medical students showed increased CK levels. From this, the researchers suggested that it is the correlation between AST, ALT and GGT that shows true liver dysfunction. Keep in mind, we can only guess that the 15 steroid users were using 17 alpha-alkylated steroids, and we do not know what the dosages that were used., but common sense tells us the results are likely relevant.

    Last but not least, a simple study done in 1996, showed the long term benefits after taking a 3 month break from steroids[7]. 16 bodybuilders using steroids were compared to 12 bodybuilders that were not. After a three-month drug withdrawal, the researchers showed that levels of liver enzymes, types not mentioned, returned to the same as the non users. Again the dosages are left to the reader’s imagination and we can only guess that the 16 steroid users were using 17 alpha-alkylated steroids.

    So what can we conclude from all of this? First off, 17 alpha-alkylated steroids are hepatotoxic in high dosages taken for a long time. On the other hand, short cycles and small dosages appear to be perfectly safe. I suggest that maximum dosages should be 500mg to 900mg per day. They should be cycled for perhaps 8 weeks at a time, and if needed a 3-month break from them should be used. Using the above-mentioned techniques, your liver can be healthy for a long time. Simply put, the hysteria surrounding “hepatoxic” steroids, is based mainly on folk lore.


    References:

    [1] Lancet 1979 Nov 24;2(8152):1120-3, Hepatic angiosarcoma associated with androgenic-anabolic steroids. Falk H, Thomas LB, Popper H, Ishak KG.

    [2] J Gastroenterol 2000;35(7):557-62, Multiple hepatic adenomas caused by long-term administration of androgenic steroids for aplastic anemia in association with familial adenomatous polyposis. Nakao A, Sakagami K, Nakata Y, Komazawa K, Amimoto T, Nakashima K, Isozaki H, Takakura N, Tanaka N.

    [3] J Pharmacol Toxicol Methods 1995 Aug;33(4):187-95, Toxic effects of anabolic-androgenic steroids in primary rat hepatic cell cultures. Welder AA, Robertson JW, Melchert RB.

    [4] Arch Toxicol 1999 Nov;73(8-9):465-72, Evaluation of acute and chronic hepatotoxic effects exerted by anabolic-androgenic steroid stanozolol in adult male rats. Boada LD, Zumbado M, Torres S, Lopez A, Diaz-Chico BN, Cabrera JJ, Luzardo OP.

    [5] Med Sci Sports Exerc. 1999 Feb;31(2):243-50, Rat liver lysosomal and mitochondrial activities are modified by anabolic-androgenic steroids. Molano F, Saborido A, Delgado J, Moran M, Megias A.

    [6] Clin J Sport Med 1999 Jan;9(1):34-9, Anabolic steroid-induced hepatotoxicity: is it overstated? Dickerman RD, Pertusi RM, Zachariah NY, Dufour DR, McConathy WJ.

    [7] Int J Sports Med 1996 Aug;17(6):429-33, Body composition, cardiovascular risk factors and liver function in long-term androgenic-anabolic steroids using bodybuilders three months after drug withdrawal. Hartgens F, Kuipers H, Wijnen JA, Keizer HA.

    Avant Labs is a division of Par Deus, Inc.
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    post edited by MaxMuscle - 2005/05/18 23:17:13

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    #2
    MaxMuscle
    Super Heavy Weight
    RE: AAS With a Little Help from AvantLabs 2005/05/18 23:09:31 (permalink)
    0
    SesaThin and Androgens
    by Par Deus

    I am going to start doing a series of mini-articles, looking at the usage of various Avant Labs products for a variety of specific goals. This is the first of these. As with my full SesaThin™ article this month, I did not get this finished in time, so this is just a teaser. IOW, they will not be THIS “mini”. They will be more like 5 or so pages vs. the one page that this is. I hope to have the full article by Monday, November 1st.

    While androgens are the unquestioned king for building muscle mass and strength, they do have their downsides. We will not get into the HPTA, legality, morality, etc. here today. Instead we will focus on visceral adipose tissue (‘roid gut), lipid profiles, heart disease and liver toxicity -- and why SesaThin™ should be a staple of any androgen cycle (particularly with methylated androgens, which are far worse than non-methylated androgens). This situation is even worse if the androgen is non-aromatizing, as estrogen exerts a protective effect on blood lipid profiles.

    It is well established in the literature and the real world, that heart disease and abdominal fat accumulation occurs far more often in males than in females. The reason for this is testosterone. And, of course, synthetic androgens are structurally similar and share the same pathways.

    Testosterone does its damage in several ways -- by increasing angiotensin II and beta receptor density, as well as by decreasing 11beta-hydroxysteroid dehydrogenase activity. All of these act in concert to increase activity of cortisol and the stress axis. And, the stress axis is intimately linked with visceral adipose tissue accumulation, elevated blood lipids, oxidative stress/inflammation and heart disease.

    SesaThinâ„¢ was tailor made by nature to protect against these pathologies. First and foremost, it decreases triglyceride (TG) formation and increases TG uptake and fatty acid oxidation, while decreasing cholesterol levels. It also just happens to be a potent anti-oxidant and anti-inflammatory (as well as inhibiting metabolism of the anti-oxidant/anti-inflammatory, vitamin E)

    In addition to these health benefits, prevention of VAT accumlation will keep your belly from sticking out past you massive chest like a Mr. Olympia contestant, thus it should be most welcomed by the aesthetically cognizant steroid user as well.

    As for the liver, SesaThin has also been found to be protective against liver damage, likely due to its anti-oxidant activity and inhibition of lipid accumulation.

    So, for your next cycle, do not forget the SesaThinâ„¢
    #3
    enrico
    Testo-Boy
    RE: AAS With a Little Help from AvantLabs 2005/05/18 23:44:38 (permalink)
    0
    grote hap om ineens door te nemen.
    morgen zullen we er eens aan beginnen
     
     
    #4
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